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This plasticity might be induced by Ca2+ influx through CP-AMPARs [56] which are expressed in the superficial dorsal horn [10] [13] and, as we show, are activated by release from SG EINs.
Firstly, Translation Factor eIF6, also demonstrated in the western blot, which can regulate translation by modulating the access of the large ribosomal subunits to the elongation cycle, binds to 60S subunits which are activated by release of eIF6 [ 48].
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If at least one node of the inner part of ( i i + 1, j j ) (lower solid arrow in Figure 16) receives a new value, the subdomain is activated by releasing the compute lock, CL ( i i + 1, j j ) ← Open.
From the point of views of function, microglia can be activated by releasing oxygen free radical, nitric oxide, protease, and inflammatory cytokines and take the cytotoxic effect.
First, due to an aberrant apoptosis and/or insufficient removal of apoptotic cells, the immune system in SLE is activated by released (modified) apoptotic debris.
It is also important to note that, in our previous work, SR Ca content was measured indirectly, from the integral of the NCX current activated by releasing Ca from the SR.
Manual drifting does allow you to use Mini-Turbos, which can be activated by releasing B (the drift button) when you see blue or orange sparks from behind your vehicle as you drift.
The fact that C-fibre stimulation in infants stimulates expression of anti-inflammatory IL-10 and IL-4 (McKelvey et al., 2015), suggests that their release is directly activated by neurotransmitter release from C fibre terminals in the dorsal horn and that the response following infant nerve injury could be a direct result of activity in damaged C fibre afferent terminals.
The biological activity of FZC18 is cryptic within full-length C18, as it is activated by proteolytic release.
The RanGTP concentration gradient surrounding mitotic chromosomes induces the formation of a gradient of spindle assembly factors (SAFs) activated by RanGTP‐induced release from inhibitory complexes of SAFs with importin β.
The intrinsic pathway is activated by mitochondrial release of apoptotic proteins secondary to genomic stress.
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