The phrase "activated by exogenous" is correct and usable in written English.
It can be used in contexts related to biology, pharmacology, or any field discussing external factors influencing a process or reaction.
Example: "The enzyme was activated by exogenous factors, leading to a significant increase in metabolic activity."
Alternatives: "triggered by external" or "stimulated by outside".
As the activation of TLRs results in its interaction with adaptor molecules and downstream signaling, leading to upregulation of genes with NF-κB elements, a NF-κB luciferase reporter system was used to determine whether the expressed recombinant goose TLR could be activated by exogenous macrophage-activating lipopeptide (MALP-2, 10 ng/ml) or Lipopolysaccharides (LPS, 50 ng/ml).
These compounds inhibit Tcf-mediated tractivatedon activated by exogenous Dvl via the biochemical antagonism.
The design of delivery vehicles that are stable in circulation but can be activated by exogenous energy sources is challenging.
Various models have focused on particular aspects of stem cell physiology including gene regulatory networks, gene expression noise and signaling cascades activated by exogenous factors.
It was provocative to determine if AMPK in ρ° BAEC could be activated by exogenous ONOO−.
Similarly, both compounds blocked JAK/STAT signaling stimulation when the pathway was activated by exogenous IL-6 treatments (Fig. 2B).
In addition to replicative senescence, human fibroblasts can undergo stress-induced premature senescence (SIPS) via activation of the MAP kinase p38 that responds to and is activated by endogenous and exogenous cellular stress (Freund et al. 2011).
The tumor suppressor protein p53 is activated by endogenous or exogenous DNA damage and other cellular stresses through its posttranslational modifications such as phosphorylation, acetylation and sumoylation [ 21].
NKT cells can be activated by endogenous and exogenous antigenic lipid and glycolipid ligands exposed at the cell surface of infected cells by non-polymorphic MHC I-like proteins from the CD1 family.
Our experiments suggest that TLR4-mediated signactivatedated by endogenous or exogenous ligands induce the production of IL-12 by macrophages, mast cells and Gr-1+ cells, which enhance IL-1β and IFN-γ production, thereby suppressing TGF-β production.
Since GPR55 can be activated by endogenous and exogenous cannabinoids we discuss in the following paragraphs the possibility that GPR55 may have a functional role in GI physiology and pathophysiology.
