Sentence examples for activated by components from inspiring English sources

Based on the theoretical "two-hit hypothesis" for the development of transfusion-related ALI wherein neutrophils are initially primed by a risk factor (i.e., sepsis) and then activated by components of transfused blood [14, 30], patients with sepsis who are exposed to RBCs may be a population at increased risk for ALI after transfusion with blood of a long storage duration.

TLR2 is activated by components of Gram-positive bacteria, while TLR4 is activated by components of Gram-negative bacteria [14].

TLR2 is activated by components of the Gram-positive cell wall, whereas TLR4 is activated in response to components of the Gram-negative cell membrane [14].

Additionally the expression of TSC22D3, a regulator of T cell receptor mediated cell death, was found higher in PP, this protein may be induced by glucocorticoids [ 43, 44] activated by components in the PP diet.

This response is activated by components of the wall of invading micro-organisms, such as lipopolysaccharide (LPS) or peptidoglycan, following the binding of these pathogen-associated molecular patterns to pattern recognition receptors, such as the Toll-like receptors (TLRs) on tissue macrophages.

Based on the theoretical "two-hit hypothesis" for the development of transfusion-related ALI wherein neutrophils are initially primed by a risk factor (i.e., sepsis) and then activated by components of transfused blood [ 14, 30], patients with sepsis who are exposed to RBCs may be a population at increased risk for ALI after transfusion with blood of a long storage duration.

TLRs are activated by various components of both bacterial and viral cell components e.g. TLR4 binds lipopolysaccharide (LPS) in gram-negative bacteria and TLR7 binds single stranded RNA (ssRNA) from viruses.

Enhanced replication of the P/V mutant in MDM previously activated by bacterial components correlated with a reduced ability to produce IFN-beta in response to virus infection, whereas IFN signaling was intact.

It has indeed been demonstrated that Toll-like receptors 2 and 4, expressed on antigen-presenting cells of the iris, choroid, and ciliary body (anterior AU) [[23]], can be activated by bacterial components (i.e., the peptidoglycans of gram-positive bacteria and the lipopolysaccharides of gram-negative bacteria) and provide the link between innate and cell-mediated immune responses [[24]].

To the left of Figure 5 there is a column with 15 neurons which will be activated by the components of the input vectors 5BLC k L, where L indicates learning.

For the small number of cluster 3 genes (15 probes), including pro-inflammatory cytokines (IL1B, IL1A, IL6), baseline expression (most likely activated by serum components) and upregulation at 4 and 24 hrs of poly(I C -stimulation were suppressed I C -stimulationcells.