Our results showed that BZcon1 was directly involved in activating the development of conidiophores, because the BZcon1 mutants lost the conidia production capacity.
This result suggested that BA treatment activates the development of arrested pistil primordia in male flowers by repressing the expression of the TS2 homolog in J. curcas, which resulted in an increase in the ratio of female to male flowers.
RANKL activates the development of pre-osteoclasts to become mature osteoclasts, while osteoprotegerin or OPG serves as a secreted receptor of RANKL (decoy receptor), resulting in inhibition of RANKL binding to its cell surface receptor and activation of osteoclast maturation.
This crosstalk can occur at a number of levels, including the type-1 tyrosine kinase growth factor family (epidermal growth factor receptor and HER2) [ 32] and the PI3K/Akt pathway [ 33] interacting directly to activate ER signalling, with evidence that these pathways become upregulated or activated during the development of endocrine resistance [ 34].
Our results are consistent with those of Jammes et al. [ 38], who found genes encoding pectin esterases and pectate lyases were activated in Arabidopsis thaliana roots after infection with Meloidogyne incognita and the cell wall loosening process was activated during the development of the giant cell as well.
Once the endothelium becomes activated during the development of sepsis, it transforms into a procoagulant, antifibrinolytic, and proadhesive state [12].
When is angiogenesis activated during the development of a tumour?
Several proto-oncogenes are highly activated during the development of this malignancy.
This result suggests that notch signaling is activated during the development of neuropathic pain.
