The phrase "activate subsequent" is correct and usable in written English.
It can be used in contexts where you want to indicate the initiation of a process or action that follows another event or action.
Example: "Once the initial setup is complete, we can activate subsequent features to enhance the user experience."
Alternatives: "trigger following" or "initiate next steps".
Once in the dendritic spine, Ng awaits for the Ca2+ increase, which is enough to release CaM to activate subsequent targets.
Toll-like receptors, which stimulated by pathogen-associated molecular patterns such as lipopolysaccharides (LPS), induces the releasing of many kinds of proinflammatory cytokines to activate subsequent immune responses.
Following binding to DNA mispairs or IDLs and undergoing ATP-hydrolysis-dependent conformational transitions, the MutSα or MutSβ complexes further recruit another heterodimeric complex of MLH1 and PMS2 (MutLα) to activate subsequent MMR steps such as the excision of the mispairs or IDLs and the resynthesis of the DNA strand [ 1, 2].
However in the current study PD 98059 did not completely abolish the stimulatory action of IL-1β, suggesting that alternative pathways, activated subsequent on tyrosine kinase activity, also play a role in the signalling of the proliferative responses to IL-1β.
In LmnaDhe/+ fibroblasts and other Lmna/LMNA mutant cells, the TRP53 pathway is activated subsequent to DNA damage, although further downstream activation of apoptosis fails [20] [22], [43].
C3b carries out several functions: It brings about lysis (bursting) of the target cell by activating subsequent steps in the cascade, leading to the formation of a ringlike structure called the membrane attack complex.
In the parlance of the DMC model, this pattern is suggestive of a late-acting reactive control mechanism, in that left IFG activates subsequent to the onset of interference, assumingly in an effort to resolve its effects.
These ion channels can be activated subsequent to either depletion of Ca2+ from internal stores or through receptor-mediated processes [ 107].
The local increase in Ca2+ within the spine allows calmodulin (CaM) to be in its Ca2+-binding conformation and activates subsequent targets.
The resultant networks were further evaluated by the recall procedure, where a CA3 unit of the top layer (with overlap of 4) is initially activated and subsequent activity propagation in CA3 units according to recurrent connections is evaluated.
15– 18 From activation of G-protein coupled receptors by adenosine, norepinephrine, bradykinin, and opioids, phosphoinositide-3-kinase/protein kisactivatedactivated with subsequent downstream activation of nitric oxide synthase and nitric oxide formation, and guanylate cyclase, protein kinase G, and protein kinase C (PKC) activation in acute ischemia.
