The phrase "activate parallel" is correct and usable in written English.
It can be used in contexts related to technology, computing, or processes where multiple tasks or functions are initiated simultaneously.
Example: "To improve efficiency, we need to activate parallel processing in the system."
Alternatives: "enable parallel" or "initiate parallel".
In contrast, our results suggest that fibronectin and PDGF-BB may activate parallel, reinforcing intracellular signaling cascades that converge in the activation of MAPK and are therefore less sensitive to MAPK inhibition.
To activate parallel fibers and their postsynaptic targets (Purkinje cells and interneurons) an epoxylite-coated tungsten microelectrode (∼5 MΩ) was placed just into the molecular layer.
Current efforts to predict drug efficacy are generally focused on genetic and transcriptional markers of pathway activation or drug binding, such as resistance mutations that sterically hinder small molecule binding or activate parallel or orthogonal signaling pathways.
Unfortunately, resistance occurs in approximately 30% of patients and the mechanisms of resistance act directly on the ER pathway and/or activate parallel pathways that block the anti-proliferative and pro-apoptotic actions of tamoxifen [ 1, 2].
All of these mutations are proposed to activate signal transduction pathways, which either belong to the wild-type kinase counterpart or, in the case of FLT3 mutation of the juxta membranous region, activate parallel signal transduction pathways that are not activated by ligand-stimulated wild-type FLT3.
Most subsequent cerebellar-learning models, however, have followed Albus in assuming that climbing fiber activity would be an error signal, and would cause synchronously activated parallel fiber inputs to be weakened.
Van Dyken, S. J. et al. Chitin activates parallel immune modules that direct distinct inflammatory responses via innate lymphoid type 2 and γδ T cells.
Marr assumed that climbing fiber input would cause synchronously activated parallel fiber inputs to be strengthened.
The results of the study suggest that HGF is highly activated, parallel to the adsorption of F/P MPs, and that HGF release is correlated to the diffusion and/or biodegradation of the F/P MPs.
Therefore, in response to matrix detachment, cells activate two parallel programs: (1) attenuation of oxidative metabolism of glucose to decrease mitochondrial ROS production and (2) enhancement of antioxidant capacity to detoxify ROS, which allow them to lower oxidative stress and prolong survival in suspension.
This suggests that even in the presence of wild-type p53 protein, DMC might activate a parallel p53-independent pathway because the p53 target genes are not as robustly turned on by DMC in spite of stabilization of the p53 protein.
