Sentence examples for activate pain from inspiring English sources

As blood flows in and fluid leaks out, the region swells, which can put pressure on surrounding nerves, causing pain; inflammatory molecules may also activate pain fibres.

The molecular associations that are common to these disorders are not known, but include shared genetic risk factors [6, 9], regulation of brain cations [10], or common receptor signaling events that activate pain [11], inflammatory [12], or oxidative [13] pathways.

In accord with this hypothesis, bacterial virulence proteins have been shown to activate pain receptors 38.

Sensory irritants activate pain sensitive receptors in the trigeminal nerves (e.g. Alarie et al. 1996; Bessac and Jordt 2008; Julius and Basbaum 2001).

However, applying placebo needles in clinical trials implies methodological problems e.g. artificial setting, loss of credibility in acupuncture experienced by patients while mechano-sensitive Aβ-fibres are used, which by itself can activate pain inhibitory systems [ 32, 33].

That is, the widespread pain in FM is sufficient to activate pain inhibitory mechanisms, and one consequence of this inhibition is reduced resting and evoked activity in the thalamus.

Drosophila Pain and Pyr function as thermosensors responding to different "hot" temperatures (The temperature thresholds for activating Pain and Pyr are 42.6 and 37.5-40°C 37.5-40°Cvely) [ 8, 10, 35], and Drespectivelyrw functions as a moist receptor for hygrosensation [ 14].

Morrison et al. [ 36] concluded that the neuronal populations in the aMCC that activate to pain and motor processing lie adjacent to another and interact during vicarious pain.

It turns out that nerve growth factor has a second role: it is released after an injury and helps activate the pain pathway.

More specifically, sustained ascending nociceptive input can activate descending pain facilitatory systems from the rostroventromedial medulla (RVM) through the release of pronociceptive excitatory neurotransmitters.

Although this has not been shown directly for the trigeminal area, animal studies showed that vagal nerve stimulation can activate descending pain inhibitory systems, probably involving projections from the NTS to the nucleus raphe magnus and the locus coeruleus, which are at the origin of serotonergic and noradrenergic descending pain inhibitory pathways [51].