The phrase "acquired deficiencies" is correct and usable in written English.
It can be used in contexts related to health, education, or skills, referring to deficiencies that have developed over time rather than being present from birth.
Example: "The study focused on the acquired deficiencies in language skills among students who had missed significant classroom instruction."
Alternatives: "developed deficiencies" or "gained deficiencies".
Congenital or acquired deficiencies of AT expose affected patients to an increased risk of developing unprovoked and recurrent thrombo-embolic diseases.
This scenario explains why hosts with genetic or acquired deficiencies in their antibacterial immune response suffer from severe mycobacterial infections.
Inherited as well as acquired deficiencies in specific DNA mismatch repair (MMR) components are associated with the development of a wide range of benign and malignant neoplasms.
Defects in adequate inhibition of complement caused by inherited or acquired deficiencies of complement inhibitors could thus be involved in development and exacerbations of SLE nephritis.
On the basis of these findings, the MN expression appears to be greatly modulated by the inherited and acquired deficiencies in host defence mechanisms against genotoxic compounds.
Most population prevalence studies have found V Leiden mutation to be roughly an order of magnitude more prevalent than protein S, protein C or antithrombin deficiencies[ 11] though it could be argued that studies based on healthy populations could over-represent the ratio of V Leiden mutation to these other defects in symptomatic patients because of the possibility of acquired deficiencies.
Part of what happens to Yunior in this story is that by the end he begins to address that socially acquired deficiency — he actually starts to gain the ability to see women.
Whether due to a primary (genetic) or secondary (acquired) deficiency, absent or diminished enzyme activity allows undigested sugars to accumulate in the lumen of the small intestine, resulting in the clinical effects of sucrase-isomaltase deficiency [12].
Bradykinin-mediated angioedema can result from many causes, including hereditary defects in C1 esterase inhibitor (C1-INH), side effects of angiotensin-converting enzyme inhibitors (ACEis), or acquired deficiency in C1-INH.
Acquired deficiency of renal Dnase1 activity is assumed to promote a progressive exposure of secondary necrotic chromatin in GBM, and a consequent development of severe nephritis [30], [31].
However, this level of protection may be compromised by inherited or acquired deficiency of these proteins.
