The phrase "acquire survival" is not commonly used in written English and may sound awkward.
It could be used in a context discussing the means or methods to ensure one's survival, particularly in extreme situations.
Example: "In the wilderness, one must learn to acquire survival skills to endure the harsh conditions."
Alternatives: "attain survival" or "secure survival".
Marina Warner: Insofar as women are underdogs, we acquire survival skills through cooperation and negotiation, are quick to sense potential friends and defenders, form a group, and develop a nose for danger and consequently manoeuvre to quarantine its carriers.
Many cancer cells acquire survival advantage during tumor progression by decreasing its sensitivity to Fas-induced apoptosis [6], [7].
During tumor progression cells can acquire survival advantages by decreasing Fas expression [31], [32], [33] and its sensitivity to Fas-induced apoptosis [7].
Therefore, it seems likely that BRCA1-mutated cells acquire survival factors that allow BRCA1-deficient tumor cells to expand.
Thus, although tumor-initiating cells/stem-like cells may be of primary importance, these cells might acquire survival advantage in gene expression via epigenetic mechanisms.
On the other hand, it may help drug-sensitive cancer cells to acquire survival signals from drug-insensitive cells and escape death during cancer treatment.
Stem cells have a greater propensity to accumulate genetic and epigenetic modifications in order to acquire a survival advantage when under selection pressure [ 12].
Therefore, the suppression of JNK1-mediated PKM2 Thr365 phosphorylation by PARP14 may represent an unexpected mechanism by which HCC cells acquire a survival advantage.
Fas expression decreases during tumor progression [31], [32], [33], and many cancer cells acquire a survival advantage by decreasing its sensitivity to Fas-induced apoptosis [7].
We hypothesise that aberrant upregulation of FOXM1B may be inducing genomic instability through a program of malignant transformation involving deregulation of cytokinesis (CEP55) and epigenetic modifications (HELLS) whereby genomically unstable cells acquire oncogenic survival advantage (Fig. 8B).
Data were evaluated to acquire cumulative survival rates according to gender, age, tobacco use, surgical procedure, bone quality, and restoration type.
