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There was improvement in esophageal acid exposure and regurgitation.
a: Normal proximal and distal esophageal acid exposure.
b: Abnormal distal esophageal acid exposure during upright position.
Esophageal acid exposure time was also decreased in the combination therapy group.
Of the 65 patients studied, 38% exhibited only bile acid reflux, with no pathologic acid exposure.
Mechanical distention, acid exposure, temperature, and osmolality-related stimuli can all induce esophageal pain.
Periconceptional folic acid exposure and risk of occurrent neural tube defects.
Acute acid exposure also appears to enhance sensitivity to mechanical distention.
Abnormal esophageal acid exposure was documented in 62% of this cohort.
Abnormal esophageal acid exposure was demonstrated in 62%, with pharyngeal reflux documented in 30%.
Nocturnal acid breakthrough: clinical significance and correlation with esophageal acid exposure.
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