The phrase "achieved muscle" is not commonly used in written English and may be unclear without context.
It could be used in contexts discussing fitness or bodybuilding, referring to muscle that has been developed or gained through training.
Example: "After months of dedicated workouts, she finally saw the results of her hard work and achieved muscle definition she had always wanted."
Alternatives: "developed muscle" or "gained muscle mass".
Signalling initiated by tumour necrosis factor (TNF), interferon-γ (IFNγ), interleukin-10 (IL-10) and insulin-like growth factor 1 (IGF1) has key roles in controlling the normal inflammatory response and myogenic response to muscle damage that is required to achieve muscle regeneration.
Muscular training is the primary mode to achieve muscle restoration, especially after traumas and injuries.
The crucial principle of this technique is to provide sufficient resistance to achieve muscle fatigue within 8 to 12 repetitions of an exercise.
In general, a training intensity of more than 60% of one's concentric one repetition maximum (1RM) is commonly considered the minimum intensity required to achieve muscle hypertrophy.
These were first used to achieve muscle specific deletion of PDK1, producing mice that die early with severe defects in cardiac function [3].
Our data suggest that electrical stimulation of the skeletal muscle with low-force generation can be beneficial in achieving muscle hypertrophy without the pain associated with high frequency electrical stimulation.
For the remaining strengthening exercises, resistance was achieved using weights, Therabands™ (rubber tubing) or springs, selecting a weight or length of tubing or spring that would achieve muscle fatigue within 10 repetitions.
Even though testosterone and IGF-1 values were in or above the normal range for healthy individuals, the patients did not achieve muscle hypertrophy after a rigorous period of high load resistance training.
It is obvious that human studies are far from the animal models, and our LFS model underscores the theory that activation of type II fibers is a key factor in achieving muscle hypertrophy without high exerted force.
This argues that the LRC pool is actively set aside into a relatively dormant state in spite of the inductive environment necessary for the proliferation and fusion of Pax7+ cells in order to achieve muscle growth.
