In order to achieve this, mCherryHi expression would need to be combined with cell surface marker staining.
To achieve this transient expression, both epigenetic mechanisms and trans-acting factors are involved.
To achieve this, the expression of a panel of markers was monitored in chick (Gallus gallus) embryos from stage 9 to stage 21 of development on consecutive sections (Fig. 1; supplementary material Fig. S1).
To achieve this, we generated expression constructs for a series of TDP-43 variants, including TDP-43WT, synthetic mutants (TDP-43SM) and ALS/FTLD-linked TDP-43MS (Fig. 1A).
However, it did not achieve this level of expression when packaged in wild-type AAV vectors and subretinally injected into mice.
On the other hand MEP and Pathway Tools hole filler represent an alternative approach that tackles the gap filling issue identifying missing genes rather than missing reactions, and these tools achieve this goal using expression data and species homology, respectively.
Tumours require a vascular supply to grow and can achieve this via the expression of pro-angiogenic growth factors, including members of the vascular endothelial growth factor (VEGF) family of ligands.
To achieve this objective, genes expression analysis was performed by qRT-PCR on total RNA isolated from entire WT plantlets treated for 24 h with increasing concentrations of NaCl.
