Sentence examples for ache p from inspiring English sources

Dichlorvos significantly reduced the kidney AChE (p < 0.001), BChE (p < 0.01) and GABA level (p < 0.01) compared to control.

Interestingly, the treatment with TL, on the other hand, restored the activities of AChE (P < 0.05).

In addition, in the LFD+VEH group, the mean levels of pTau (P < 0.001), AβPP (P < 0001), and AChE (P < 0.05) were significantly lower than in the chronic HFD-fed group.

Early limited exposure to NDEA significantly reduced the mean levels of pTau (P < 0.001), AβPP (P < 0.01), ChAT (P < 0.05), and AChE (P < 0.01) relative to the levels measured in LFD+VEH treated controls.

Chronic HFD feeding alone (HFD+VEH) significantly reduced pTau (P < 0.01) and ChAT (P < 0.001) relative to control, whereas early NDEA exposure plus later chronic HFD feeding significantly reduced Tau (P < 0.001), pTau (P < 0.001), ChAT (P < 0.001), and AChE (P < 0.001) relative to control and chronic HFD fed rats.

Thioflavin T (1), the known AChE P-site inhibitor, was observed to also inhibit BuChE with comparable potency (Table 1).

Since inhibitor binding site competition analysis and mutant studies were successful in mapping AChE P-site components, a similar approach was made to probe the BuChE P-site.

X-ray crystallography studies corroborate a kinetic approach that determined binding site competition between these inhibitors, thereby helping to define details of the AChE P-site.

Another fluorescent inhibitor probe, propidium (2), also interacts with the AChE P-site, but it is thought to be able to enter the larger active site gorge of BuChE and bind closer to the catalytic triad.

All variable were significant at P < .0001 except for shortness of breath (P =0.0300), body aches (P = 0.002), and asthma (P = 0.0002).

Relapse patient records were 2.7 times more likely to report aches (p = 0.004) and 2.8 times more likely to note lymphadenopathy (p = 0.04).