Juveniles that encountered improving conditions during the larval stage compensated for delayed growth by accelerating subsequent growth, and thus showed no evidence of a reduced reproductive rate.
Evolution via dosage effects can thus accelerate subsequent adaptation and innovation by increasing the evolvability of organisms.
VRN-1 downregulates the floral repressor VRN-2, and allows long-day induction of the floral activator VRN-3 to accelerate subsequent stages of floral development [ 17, 19].
Similarly, de novo learning of an alternation task was facilitated by CA3 NMDARs; this experience accelerated subsequent learning of related tasks, independent of CA3 NMDARs, consistent with a schema-based learning.
The presence of CA3 NMDARs facilitated the de novo learning of the alternation task; this prior experience accelerated subsequent learning of a related alternation task in a CA3 NMDAR-independent manner, consistent with principles of a schema-based learning (Bannerman et al., 1995; Tse et al., 2007; McKenzie et al., 2013).
VRN1 is central in the vernalization pathway [ 18] as it down regulates the floral repressor VERNALIZATION2 (VRN2) but also interacts with other flowering pathways, allowing long-day induction of the floral activator FLOWERING LOCUS T-like 1 (FT1) to accelerate subsequent stages of floral development [ 4, 19- 22].
In such a situation the LP optimization can play the additional role of filtering the initial set of genomes: for example, in the Pseudomonas ndLP analysis we reduced the candidate set of 'interesting' genomes from 849 to 203, which would accelerate subsequent steps of orthologous set inference, sequence alignment and phylogenetic analysis.
Because pre-incubation of Sly1 at 30°C did not accelerate subsequent Sly1 binding to Sec17-SNARE complexes, we suggest that elevated temperature increases the frequency of interconversion between the putative ground and binding-active conformations, rather than by stabilizing an activated conformation.
PPP1R2 is also phosphorylated at the residue Ser86 by casein kinase 2 (CK2) that accelerates the subsequent phosphorylation at Thr72 by GSK3 [ 16].
Exposure of cells to IL-1 in the presence of NOS inhibitors potentiates or accelerates the subsequent post-transcriptional down-regulation of CYP2B proteins in response to pharmacologically or cellularly generated NO.
