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However, in IIN, other than reduced visual acuity and an abnormal optokinetic response (9, 10), no overt ocular abnormality has been described.
This functional brain abnormality has been detected in EPs for virtually all sensory modalities [3].
A basis for such abnormality has been identified in a defective ability to habituate to repetitive sensorial stimulation.
This abnormality has been reported in the majority of EHEs at various anatomical sites, while it is absent in other epithelioid vascular tumors [11 13].
Assuming the abnormality has been adequately sampled, there is no current evidence to suggest that there is a role for specific follow-up in biopsy-proven benign disease.
Unlike myxomatous degeneration in Marfan syndrome, which has been reported to result from a mutation in the gene that codes for the extracellular structural protein fibrillin, no specific molecular abnormality has been documented to be the underlying cause of myxomatous degeneration in mitral valve prolapse syndrome (MVPS).
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Finding more abnormalities has been a great strategy for our industry.
However, the mechanism underlying these abnormalities has been less studied.
A large number of endocrine abnormalities has been described in patients with primary headaches [71, 72].
No explanation for these abnormalities has been suggested.
The presence of psychiatric abnormalities has been substantiated by several studies.
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