The phrase "abnormalities in the generation" is correct and usable in written English.
It can be used in contexts discussing issues or irregularities related to the process of generation, such as in biological, technological, or data generation scenarios.
Example: "The study revealed several abnormalities in the generation of neural cells, indicating potential developmental issues."
Alternatives: "irregularities in the generation" or "anomalies in the generation".
Further analysis revealed defects in lung, gastrointestinal tract, and heart in Fendrr −/− neonates, whereas linc Brn1b −/− mutants displayed distinct abnormalities in the generation of upper layer II IV neurons in the neocortex.
Abnormalities in the generation of Aβ are considered as the causal factor for the familial forms of early-onset AD, whereas poly-morphisms of the genes encoding for ApoE, clusterin and complement proteins are the genetic risk factors for late-onset AD.
Many of the abnormalities in the F2 generation were identical or homologous to those in the F1 generation.
We did not observe a latitude effect in the abnormalities in the P generation (r = 0.28, df = 8, p = 0.44) and the F1 generation (r = −0.0054, df = 6, p = 0.99) from the May samples, although we admit that relatively small number of sampling localities might require a caution.
For example, in laboratory experiments, nonylphenol caused poor survival in the subsequent generation of the Pacific oyster, Crassostrea gigas (Nice et al. 2003), and octylphenol caused developmental abnormalities in the subsequent generation of Japanese medaka, Oryzias latipes (Gray et al. 1999c).
Mice with visible abnormalities in the third generation (G3) were kept for further analysis.
Heart disease leads to abnormalities in the force-generation machinery, contractility rate and contraction and relaxation velocities.
Because mutations were introduced continuously because of the continuous irradiation, the mutations accumulated, causing a further increase in the mortality and abnormalities in the later generations.
The abnormalities in the F1 and F2 generations were more severe than in the P generation and were more numerous than in the laboratory-reared normal population.
The relevance of this is underscored by clinical findings that IBD patients have an increased risk of thromboembolic complications and display abnormalities in thrombin generation, platelet activation and function [52, 53].
The ATP concentration in sunitinib treated cardiomyocytes is significantly reduced, which corroborates abnormalities in energy generation.
