Similar increases in amygdala activity were observed in response to sad faces in remitted subjects with MDD (Neumeister et al. 2006a), suggesting that this abnormality is trait-like in an MDD subgroup.
The cascade of events hypothesized above is summarized in Fig. 2. Studies in twins, who have been raised apart and together (54), have documented that body fat distribution and metabolic abnormalities are a clustering genetic trait.
Some congenital cardiac abnormalities are inherited and may be transmitted as autosomal or sex-linked traits.
All these abnormalities are associated with GERD.
The causes of these abnormalities are complex.
These circadian abnormalities are also observed during stable phases of normal mood (euthymia) and are thus considered to be a 'trait' of BD.
On the web, no such anatomic abnormalities are required.
All newly detected abnormalities were then grouped by trait, and further subdivided into affected region including: gait (antalgic, abnormal stride length), arms (fingers and hand, wrist, elbow, shoulder), legs (toes and feet, ankle, knee, hip, other), and spine (scoliosis, lordosis, kyphosis, decreased range of motion, other).
Magnetic resonance imaging (MRI) of OA and other arthritides share many pathological traits, the most prominent abnormalities being synovitis, joint effusion and bone marrow lesions (BMLs) [ 7, 8].
No abnormalities were found.
Several abnormalities were observed.
