The phrase "abnormal vision" is correct and usable in written English.
It can be used in medical or descriptive contexts to refer to vision that deviates from the norm or typical range.
Example: "The patient was diagnosed with abnormal vision, which required further examination by a specialist."
Alternatives: "impaired vision" or "vision impairment."
Symptoms include migraine, abnormal vision, stroke-like signs, seizures, and abnormalities of consciousness and mental status.
Symptoms may include headache, abnormal vision, any of the symptoms of stroke such as weakness of the face and limbs on one side of the body, and seizures.
This RBP belongs to the ELAV (Embryonic Lethal Abnormal Vision) family of proteins [13].
Nestin MO did not influence Elavl3/4 (Embryonic lethal, abnormal vision, Drosophila-like 3/4) (a neuronal marker), or otx2 (a midbrain neuronal marker), but severely perturbed cranial motor nerve development and axon distribution.
A current model proposes that a balance between the antagonistic activities of MBNL1 and polypyrimidine tract binding protein (PTB), which promote exclusion of exon 5, and CUG binding protein 1 (CUG-BP1) and embryonic lethal abnormal vision type RNA binding protein 3 (ETR-3), which promote exon 5 inclusion, instructs the spliceosome to retain or not exon 5 in mature transcripts [10].
Children with abnormal vision or ocular signs were then further examined by pediatric ophthalmologists.
Speaking to a forensic psychiatrist, Dr Tim Rogers, in Pentonville prison, he explained as a child in the DRC he saw rats and other "abnormal visions" and was isolated as a result.
The ELAV (embryonic lethal, abnormal vision -like 1 (HuR) protein has vision -likes in different cellular mechanisms including apoptosis [ 34, 35], oncogenesis [ 36], cellular responses to stress [ 37], and the stabilization of several cellular mRNAs [ 38].
We generated Drosophila larvae expressing these proteins in all neurons, under control of the ELAV (embryonic lethal abnormal vision -Gal4 (galactose-responsivision -Gal4tion factor) driver, and whole mount brains from these animals were stained with anti-active Caspase-3 antibodies (Supplementary Figure S4).
Patients with SCA11 exhibit symptoms of movement disorder indicative of progressive cerebellar ataxia and abnormal visions, but their life expectancy remains similar to that of the general population [ 22].
Patients diagnosed with RCD initially complain of night blindness due to rod dysfunction followed by progressive visual field constriction, abnormal color vision, and eventually loss of central vision due to cone photoreceptor involvement [ 1].
