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PW has been applied to display the normal and abnormal "signature" waveforms that are specific to each vessel.
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Furthermore, data from orbits showing abnormal signatures were discarded.
The signatures have been determined as a vital pre-requisite for the identification of abnormal signatures attributable to shaft and gearbox defects.
The spheroid abnormal karyotype signature included loss of copy number on chromosome 5, 6, 8, and 13, and gain of copy number on chromosomes 1, 5, and 17.
In concert, this abnormal signaling signature affects endothelial cell migration and adherens junction dissolution in response to VEGF [ 14, 16, 17], explaining the vascular dysfunction in vivo.
The abnormal IRF5 transcriptional signature was determined by IRF5-SLE risk haplotype [27].
These findings may shed light on the abnormal gene expression signature and leukemic cell traits observed in ATL.
Our results are consistent with the hypothesis that brain regions with relative protection from AD pathology will also tend to show a less abnormal gene expression signature at baseline.
The measurements performed on different pathological conditions clearly indicate abnormal and characteristic signatures of the vasculature (see Table 1 >).
The S1-signature reflected abnormal activation of the WNT signaling pathway, the S2-signature was described by the proliferation pathway as well as MYC and AKT activations, and the S3-signature was associated with hepatocyte differentiation.
Somewhat surprisingly then, there is little consensus as to which specific microstructural features provide a signature of abnormal growth.
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