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An interesting phenomenon with abnormal capacity increasing appears which could be ascribed to the continual enhancement of interface Li-storage.
The electrochemical behavior of cobalt terephthalate (CoTP) as the organic anode was initially carried out in K-ion battery, with the main intention to test whether the abnormal capacity enhancement of CoTP previously occurring in Li-ion battery could continuously happen in the different K-ion battery system.
an abnormal capacity to pass through the blood-brain barrier with consequent meningeal leukemia, as frequently seen in Ph+-ALL [ 21].
These results confirm that the enhanced expression of NFκB1 mRNA in RA bone marrow CD34+ cells led to their abnormal capacity to differentiate into fibroblast-like cells producing MMP-1 upon stimulation with SCF, GM-CSF and TNF-α without affecting cell viability or proliferation.
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Glymour [1994] asked the reliabilist question whether there are inference methods that are guaranteed to eventually settle on a true theory of mental organization, given exhaustive evidence about normal and abnormal capacities and reactions.
Since TAK1 regulates keratinocyte function [12], there is a possibility that hair follicle defects in TAK1-deficient mice might be attributed to such abnormal capacities of keratinocytes rather than reflecting the specific function of TAK1 in hair follicle regulation.
In this regard, we demonstrated that RA bone marrow CD34+ cells have abnormal capacities to respond to TNF-α and to differentiate into fibroblast-like cells producing MMP-1 [ 2].
Bone marrow CD34+ cells from rheumatoid arthritis (RA) patients have abnormal capacities to respond to tumor necrosis factor (TNF -α and TNF -αferentiand into fibroblast-like cells prodifferentiate metalloproteintoe (MMP)-1.
Of note, we have recently demonstrated that bone marrow CD34+ cells from RA patients have abnormal capacities to respond to tumor necrosis factor (TNF -α and TNF -αferentiand into fibroblast-like cells prodifferentiate suggestintothat bone marrow CD34+ progenitor cells might generate type B synoviocytes and thus could play an important role in the pathogenesis of RA [ 2].
Using logistic regression, individuals with an abnormal lung capacity result were compared to individuals with normal lung capacity results.
An abnormal diluting capacity can be attributed to several factors.
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