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To explore the potential roles of repetitive elements in the evolution from constitutive splicing to alternative splicing, we have performed a wide analysis on SINEs, LINEs, LTRs, DNAs, LCRs and simple repeats (Table 8).
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We draw on a wider analysis of health sector responses to IPV in Malawi, which included policy analysis and the perceptions of these stakeholders on definitions, causes, and responses to IPV.
To further exemplify this feature, we also performed a genome-wide analysis on a 109 kb long ENCODE region [ENm011; chr11:1858751-1968592; (Birney et al., 2007)] that includes six genes coding for, at least, 11 transcripts, with their corresponding intergenic regions.
They performed a genome-wide analysis on 13 family members' DNA and two carriers who did not show signs of the syndrome and were immediately drawn to a region on the long arm of chromosome 5.
Recently, Paterson et al. [ 48] carried out a genome-wide analysis on repeated measures of HbA1c from the DCCT with the aim to identify genetic loci underlying glycemic control in individuals with type 1 diabetes.
Measurements of these parameters can be found in a recent genome-wide analysis on common gene expression strategies in yeast [23].
Overall our results are in line with both an independent study of four different chromatin regions [8] and a recent genome-wide analysis on nucleosome positioning [38] that have not detected any effect of htz1Δ on chromatin structure.
In this study, we conducted a comprehensive genome-wide analysis on the characterization of human chromosomal material exchange with regard to the chromosome translocations.
In this study, we performed a more genome-wide analysis on the role of WNT signaling in childhood T-ALL, also focusing on several key components of this pathway.
Furthermore, from their genome-wide analysis on an (B6xC3H/HeJ F2 intercross with a deficiency in apolipoprotein E, Logsdon et al concluded that Zfp69 variants are associated with body weight, blood glucose and cholesterol levels [ 20].
To test this hypothesis, we performed genome-wide analysis on a cohort of 20 unrelated adults diagnosed with T1D and 20 unrelated control (Ctrl) subjects to identify CNVs either enriched or depleted in the T1D cohort compared to Ctrl.
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