Sentence examples for a subsequent regulation of from inspiring English sources
Furthermore, in Arabidopsis, an over expression of miR156 determines a reduced SPL genes activity and, by a subsequent regulation of several genes, lead to an accumulation of anthocyanins [ 84], likely connected to DFR gene activation [ 85], a flavonoid biosynthetic gene.
Finally, while the data does not definitively show that the aberrant acetylation a subsequent regulation of MnSOD is causative in these pathological processes, these results do make a strong potential scientific argument for altered regulation of MnSOD.
There is good evidence that increased activation of the transcription factor NF-κB with a subsequent regulation of pro-inflammatory mediators plays a crucial role in the development of organ dysfunction occurring in sepsis (Abraham and Singer, 2007).
With this statement, the National Rifle Association and gun enthusiasts alike cheered as the Second Amendment was resuscitated into a new obstacle for subsequent regulation of guns.
As AR plays an essential role for androgen responses and subsequent regulation of steroidogenesis, our model advances the work of Shoemaker et al. [ 49] by simulating AR-related signalling pathways.
Activation of PPAR nuclear receptors and subsequent regulation of specific target genes elicit a complex series of inflammatory responses via transactivation or transrepression.
Subsequent regulation of gene activity is governed by a dual regulatory strategy utilizing two drugs, Tamoxifen and Doxycycline.
This leads to a pressing need for appropriate hazard and risk assessment, and subsequent regulation of these new and emerging nanosubstances.
Here, we studied the effect of cyclosporine on HNF4α and various RAS components to better understand cyclosporine induced hypertension and proposed HNF4α dysfunction and subsequent regulation of AGTR1, ACE, and ACE2 as a molecular rational.
Thus, the Lyn Cbp pathway and subsequent regulation of Lyn signaling and cell morphology involves a dynamic and complex series of interactions.
In addition, TSC22D4 overexpression significantly reduced the activity of a FAS reporter gene in Hepa1c1 hepatocytes in a dose-dependent manner (Supporting Information Fig S5D), further indicating that TSC22D4 represents a critical transcriptional checkpoint for the lipogenic genetic pathway and the subsequent regulation of serum TG homeostasis via control of hepatic VLDL release.
