We attempted to minimize confounding by using a robust propensity-matching algorithm based on potential confounders at the hospital and patient level.
The limb chondrogenic regulatory system operating in the presence of a gradient has an inherent, robust propensity to form limb-like skeletal structures.
This is the first population-based multicenter cohort study in patients with underlying cardiovascular disease and no pre-existing cancer diagnosis which used robust propensity score modeling to adjust for baseline imbalances and which accounted for competing risks.
Additionally, robust propensity score modeling was used to help minimize potential confounding, and competing risk methods were used given that competing risks are of notable concern due to the age and prevalence of comorbidities in this particular patient population.
In our Drosophila model of Aβ toxicity we have demonstrated a robust correlation between the propensity of an Aβ peptide variant to form oligomeric aggregates and its in vivo toxicity, as estimated by longevity and locomotor phenotypes (Luheshi et al., 2007).
While our results allow us to conclude that degree alone is a robust indicator for a proteins propensity to be evolutionary conserved, the inherent topological robustness of locally clustered links emphasizes the emergent role of cohesive areas [ 26] as mediators of evolutionary information.
Comparison of the results from this approach with experimental observations indicates that it provides a robust means of predicting the adhesion propensity.
This study improved on previous methods by excluding data for 6 months after first secure email use, comprehensively adjusting for baseline utilisation, deriving propensity scores from a robust set of independent variables, and examining clinical services utilisation 7 18 months after the index date.
Propensity score matching is a robust means of controlling for observed confounding in observational data [ 15].
The inclusion of a robust array of independent variables in the propensity score matching model is also a strength.
This study is the first to specifically identify possible nuclei which underpin the propensity of mice to exhibit a robust reinstatement of a previously extinguished CPP to cocaine.
