Sentence examples for a remarkable induction of from inspiring English sources

Egr-1 was activated by various growth factors including PDGF, and we also found a remarkable induction of Egr-1 by PDGF in RASMCs (data not shown).

Quantitative assay for apoptosis showed a remarkable induction of apoptosis in KM20C (4.2%) and SC-6 (3.5%), which were relatively sensitive to UFT.

Beside autophagy activation, a remarkable induction of Gadd34 and the slight but delayed activation of proapoptotic CHOP/Gadd153 could also be observed upon metyrapone addition (at concentrations >50  μM) confirming ER stress).

24, 25 Using this NO delivery system, we found a remarkable induction of apoptosis in tumor cells, not only in in vitro studies, but in in vivo solid tumor models.

NR delayed the development of morphological hallmarks and ultrastructural changes in mitochondria and resulted in a remarkable induction of mitochondrial biogenesis and oxidative metabolism, with an increase in mitochondrial mass, mtDNA, and respiratory chain protein amounts in the muscle.

Initiation of wandering behavior, when larvae crawl out of the food in search of a dry place to pupariate around 108 h after egg laying (AEL), or starvation before this time results in a remarkable induction of autophagy in polyploid tissues, but not in diploid cells.

In particular, both genes exhibited a remarkable induction post-fertilization, in 1 3 DAF and 3 5 DAF seeds in Caresse (approximately 8 fold) and a decline at the later stages of seed development.

Additionally, remarkable induction of HvDME was evidenced in response to drought treatment in a drought-tolerant barley cultivar.

The SiLEA14 promoter mediated remarkable induction of β-glucuronidase (GUS) expression in transgenic Arabidopsis under various stresses.

31 The remarkable induction of CTSE expression in BE intestinal metaplasia and dysplasia, together with the significant down-regulation in EAC tissues, suggests a possible role for CTSE in the Barrett disease spectrum.

The remarkable induction of the genes of sulfur metabolism in the cspC mutant indicates that the cells are actively increasing the input of sulfate and that it is probably being funneled to the methionine biosynthesis pathway.