A recent observation indicates the protein kinase CK2 as a crucial factor in aggresome assembly and clearance.
Recent observation indicates that regulation of acetylation status mediates proteolytic function in diseased myocardium from human and animal models [ 16].
An unexpected recent observation indicates that these two signaling pathways may be functionally linked by coordinated metabolism in the nervous system (Nomura et al., 2008).
Our recent observation indicates that P and MPA promote T47-D cell horizontal migration through a complex cascade requiring PR, G proteins, MAPK, PI3K and the Rho-associated kinase, ROCK-2 (X.D. Fu et al, submitted).
This idea fully agree with recent observations, indicating an advantage of convective over pure diffusive strategies for patient survival.
These findings are in accordance with recent observations indicating that, under some experimental conditions, caspase-8 may induce some cytotoxicity independently of its classical caspase activity [24].
This result was unexpected given our recent observations indicating that CTT1 expression is up-regulated in zinc-deficient cells and may be a direct target of Zap1 regulation [14].
This interaction is particularly interesting in the light of recent observations indicating that in NO-deficiency other prostanoids (e.g. prostacyclin) may also induce vascular smooth muscle effects via activation of the thromboxane (TP) receptor [18] and cerebrovascular expression of TP-receptors is enhanced after SAH [19].
Recent observations indicating that fat tissue is also associated with immune responses also suggest a link between obesity and gut inflammation [ 19].
This is in line with recent observations indicating that excitatory synapses onto CA1 pyramidal cells, during stimulation at increased frequency are largely dependent on fast refilling of synaptic vesicles, rather than mobilization of reserve vesicles (Ertunc et al. 2007).
