Exact(5)
These results suggest that compound A protects ARPE-19 cells against oxidative stress, in part, by enhancing several anti-oxidant defense mechanisms.
Recent results suggested that enhanced gelsolin expression is an important mechanism by which histone deacetylase inhibitor trichostatin A protects against ischemic brain injury [ 40].
Chen et al. reported that biochanin A protects dopaminergic neurons against LPS-induced damage through inhibition of microglia activation and generation of proinflammatory factors [ 19].
This is a major cause of reperfusion injury [ 12, 34] and inhibition of mPTP opening with CsA or sanglifehrin A protects hearts from reperfusion injury in a variety of settings, including isolated perfused hearts, animal models and patients undergoing angioplasty [ 12, 34, 35].
In contrast, Diaz et al. [ 10] in a model of I/R of rabbit cardiomyocytes in cell culture have found that cyclosporine A protects against cardiac myocyte necrosis by induction of chloride anions efflux from myocytes enhancing their cell-volume regulatory response without changes in transmembrane mitochondrial gradient.
Similar(54)
Thus, VEGF-A protects neurons from environmental stress and apoptosis and can directly stimulate neurogenesis and axonal regeneration [10], [31].
Investigation of the molecular mechanisms by which Roc-A protects normal cells from chemotherapy-induced cytotoxicity revealed that p53 is a key factor in Roc-A-mediated protection.
Apart from preventing calcium and phosphate precipitation in the serum, fetuin-A protects from arterial media calcification by inhibiting VSMC apoptosis and preventing basic calcium particle nucleation in the extracellular matrix [ 7].
We further investigated the molecular mechanisms by which Roc-A protects normal cells from DNA damage-induced cell death and revealed that the transcription factor p53 is essential for this protection.
Modulation of lymphocyte migration through targeting of SELL and CCR7 is proposed to facilitate HIV-1 immune avoidance (Ramirez et al., 2014; Vassena et al., 2015), and downregulation of NTB-A protects HIV-infected cells from NK cell lysis (Shah et al., 2010).
This is now a protected mural.
Write better and faster with AI suggestions while staying true to your unique style.
Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com