Sentence examples for a prior study shows from inspiring English sources

A prior study shows that the N-terminus of Dvl1 (K5, 20, 34, 46, 50, 60 and 69 on the DIX domain) is modified by K63-linked polyubiquitination, which requires DIX-domain mediated polymerization of Dvl [ 79].

Consistent with the notion that p38 and phosphorylation of GSK3β at Ser9 may play a role in Wnt signaling, a prior study shows that p38 MAPK is activated upon Wnt3a stimulation and is crucial for Wnt3a-induced accumulation of β-catenin through inhibiting GSK3β a activity by inducing its phosphorylation at Ser9 [ 158].

A prior study showed a spontaneous and transforming growth factor β (TGF-β -induced EndMTGF-β -induced endoTGF-β -inducedisolatEndMTom caveolin-1 knockout mice compulmonarythendothelialpe littermates (Li et al., 2013).

These regions were selected based on a prior study showing that occlusions of these segments were more likely to be associated with larger strokes [18] and based on the likelihood that proximal occlusions in these locations could be readily identified by physicians with minimal training in interpreting CTAs.

These data thus corroborate and extend a prior study showing 20% MAP DNA positivity in non-IBD subjects.

This finding is consistent with a prior study showing reduced levels of CD86 mRNA in lethal pediatric septic shock [27].

Similar adverse effects were observed in a prior study showing pro-oxidant activity for ascorbate (vitamin C), and trolox (a water soluble derivative of vitamin E) [1].

A prior study showed that in BEAS-2B cells exposed to FHA-coated wells, the NF-κB pathway is activated in an RGD motif-dependent manner [23].

For example, a prior study showed that major depression was correlated with less sexual activity, while dysthymic disorder was associated with increased unprotected sex [53].

Our findings are consistent with a prior study showing nuclear (active) β-catenin staining to be prognostic in colon cancer,[24] and highlight a role of Wnt signaling in colon cancer progression and liver metastasis.

Indeed, a prior study showed that hypothalamic deactivation after oral glucose administration was markedly attenuated in obese subjects [47] and our findings further suggest abnormal hypothalamic sensitivity to vagal stimulation in obesity.