Although no previous study uncovered the "Section Newport II" relationships (Newport + Muenchen + Litchfield), the lineage is very strongly supported in our ML tree and is also congruent with antigen characters: all have O 8 groups and 1,2 phase-2 flagellar antigens.
Previous studies uncovered a nonsense mutation in the gene encoding the deubiquitylating enzyme Ubp6 as conferring improved proliferation (faster growth rate) properties to a subset of disomic yeast strains (Torres et al., 2010), implicating proteasome function in contributing to protein homeostasis in aneuploid cells.
Several previous studies uncovered the T cell immunoregulatory properties of PP14, and defined key aspects of its mode of action.
Our previous studies uncovered critical roles for the small phosphoprotein α-endosulfine (Endos) in both oocyte meiotic maturation and early embryonic mitoses in Drosophila (Von Stetina et al. 2008).
Previous studies uncovered that IL-11 stimulates tumor cell proliferation and invasion via its downstream signaling cascade in colorectal adenocarcinoma 10 and that IL-11 overexpression is correlated with poor clinical outcomes in gastric cancer.
In a previous study [ 23], we uncovered a key enzyme, GLUCOSE-6-PHOSPHATE DEHYDROGENASE (G6PD1), that appears to play a significant role in fitness compensation.
A previous population phenotype study, uncovered very large diversity in the levels to which environmental and clinical isolates of C. jejuni invaded cultured intestinal epithelial cells in vitro [ 15].
Comparisons were made to a previous study, conducted by Pierce and Rey, 2013 [ 47], in the susceptible Arabidopsis-SACMV pathosystem, and results uncovered similar and different global patterns or trends in differentially expressed genes between the two hosts.
This was an interesting phenotype to uncover in the in vitro-produced cells but was also seen in a previous study (Kimura et al., 2006).
