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The proteins involved in drug uptake in breast cancer tissue have shown to play an active role in drug resistance [1], and we have in a previous study discovered glycoproteins involved in response to tamoxifen, distinguishing recurring from non-recurring breast cancer patients [2].
For example, a previous study discovered 5 mutational signatures in 21 breast cancer samples [ 18].
In the human HOXA locus, a previous study discovered extensive MLL1 binding events to a transcriptionally active chromatin domain [ 47].
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For example, mutation in SLIT and NTRK-like 1 (SLITRK1) can cause TS, and though there are other examples, each only accounts for a small fraction of cases [ 8, 9] Notably, our previous study discovered a set of specific alternatively spliced genes that differentiate TS from controls, suggesting that there may be a shared molecular pathophysiology common to many subjects with TS [ 10].
Our previous study discovered that A. culicicola harbors three plasmids, which we designated as pAc3249A, pAc3249B and pAc3249C.
(36) Our previous study discovered that NPC cells presented with typical morphorlogical and biomolecular changes of EMT during exposure to irradiation.
For instance, previous studies discovered that the deepest branching lineages within the bacterial and archaeal domains are thermophilic [ 1].
Previous studies discovered thousands of miRNAs and numerous miRNA target genes mainly through computation and prediction methods which produced high rates of false positive prediction.
Fortunately, previous studies discovered that the estimated coefficients of Q remained nearly unchanged when near-optimal phylogenetic trees and rate parameters were used [[ 10], and references therein].
Previous studies discovered functional similarities between btd and some vertebrate Sp genes, but could not confidently identify a genuine btd orthologue in vertebrates [ 13, 15, 25], and it had been proposed that the btd gene might be the result of a recent gene duplication when another Sp-family gene, D-Sp1, in the vicinity of btd was discovered in D. melanogaster [ 8, 20].
In a previous study, we discovered a decrease of plasma levels of carnosine dipeptidase 1 (CNDP1) in association with aggressive prostate cancer.
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