Most live piglets appeared normal at birth and there was no obvious difference in appearance between the cloned and natural bred piglets.
These piglets appeared normal at birth and were observed to have normal growth 8 months later.
Weight of the piglets appeared relatively similar between groups: the mean weight in the E group was 36.5 kg (range: 36.0 to 37.3 kg) and the mean weight in the C group was 38.2 kg (range: 34.6.6 to 40.0 kg); however, we noticed a significant difference between both groups (Wilcoxon test, P = 0.047).
Furthermore, diarrhoeic piglets appear to suffer from an imbalanced gut microbiota, in which bacteria regarded as beneficial are diminished, which particularly accounts for L. acidophilus.
As the location of the ampulla of the thoracic duct was consistent in most of the piglets, Large White piglets appear to be suitable for the development of a thoracic duct cannulation model without thoracotomy.
The piglets appear to follow mom's lead, say the researchers, trusting in her wisdom.
Male and female piglets appear susceptible to PCS in similar proportion [ 1, 5, 6].
Focussing on the highly increased IL-6 levels in LP and HP offspring 3 h after LPS injection, these piglets appear to be more susceptible to bacterial endotoxin exposure with an increased risk of an acute systemic inflammatory response syndrome [ 58].
Birth weight is the most determinant factor for piglet survival with direct impact on thermoregulatory capacity and growth; piglet vitality is also correlated with survival and growth and is strongly influenced by the degree of intra-partum hypoxia suffered by the piglet; additionally, piglet PWM appears to be sex-biased, with males showing greater susceptibility to causal mortality factors.
The apparent discrepancy between these studies may stem from the use of different models; indeed, the piglet model appears as more physiological and closer to the human case than the rodent model, which may account for a different spatio-temporal evolution of damage.
