Sentence examples for a partial gain from inspiring English sources

CT60 cells have only a partial gain of function mutation in SCAP [40], which allows them to exhibit, at least to some degree, cholesterol-regulatory homeostatic mechanisms.

The mutant CT60 cells [40], with premature translational termination of the NPC1 protein, are derived from 25RA, a CHO cell line with a partial gain of function mutation in the SREBP (sterol regulatory element binding protein) cleavage-activating protein (SCAP) [41].

Mouse IFT mutants have paradoxical phenotypes suggesting both a partial gain and partial loss of Hh function in different organs.

Silencing VE-cadherin in Ep5ExTu cells, in contrast, led to only a partial gain of the epithelial morphology and was not associated with re-induction of E-cadherin (see Additional file 5).

(Remember, compromise can be described as a partial gain and loss on both sides. Do your best to make the gain on each side a fulfilling one. If both parties are actively trying to solve the problem, it will dissipate much of the emotional aspect of the argument).

Cytogenetic analysis (array-comparative genomic hybridisation or array-CGH) was also performed, revealing a complete gain of the entire chromosome 20 and a partial amplification and gain of chromosome 6p, but without monosomy 3 or 8q gains.

a Partial total gain (pTG): hatched area/blue frame; total gain (TG): blue area.

These data suggest that loss of methylation is a feature of prostate cancer, and partial gain of methylation (presumably at promoters of specific genes) is associated with clinical outcome and is measurable using whole-cell assays.

Partial gain of chromosome arm 17q is an abundant aberrancy in various cancer types such as lung and prostate cancer with a prominent occurrence and prognostic significance in neuroblastoma – one of the most common embryonic tumors.

There are several ways to structure the 1031 exchange that can include taking partial gain ("boot") out of the sale or trade.

Partial gain of chromosome arm 17q is the most abundant genetic alteration in neuroblastoma and constitutes the strongest genetic factor for adverse prognosis [12].