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In order to directly compare the epigenetic profiles of extra-embryonic stem cell lines with those of pluripotent cell lines, we initially assessed the replication timing of a panel of developmental genes in OS25, B1 and IM8A1 cell lines.
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To investigate chromatin bivalency and interplay between PRCs and RNAPII, we first looked genome-wide at the association of PRCs with the unusual RNAPII variant (S5p+S2p−8WG16−) previously identified in a panel of PRC-target developmental genes (Stock et al., 2007).
Both these issues have been extensively considered (studying both published and unpublished work) on three separate occasions by a panel of independent experts (including developmental biologists, IVF experts and clinical geneticists).
These were selected by a panel of experts in risk communication, developmental psychology and genetic communication.
In a comprehensive review of developmental studies involving MeHg, a panel of experts recommended a more consistent use of the same endpoints across studies to facilitate comparisons.
The RNA was extracted from a panel of eight samples that represented a variety of tissues and developmental stages.
Using a panel of well-defined human teratogens, biomarkers were identified that may be suited to predict developmental toxicity.
To study the developmental effects of CFC mutant alleles in vivo, we have expressed a panel of 28 BRAF and MEK alleles in zebrafish embryos to assess the function of human disease alleles and available chemical inhibitors of this pathway.
For example, analysis of a panel of more than 200 miRNAs from 334 chronic lymphocytic leukemia samples indicated that miRNA profiles reflected the developmental lineage and differentiation status of this neoplasm [20].
Also, a panel, of "experts".
A panel of thirty LCL was used.
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CEO of Professional Science Editing for Scientists @ prosciediting.com