Sentence examples for a panel of aggressive from inspiring English sources

Cytostatic concentrations of mifepristone induced alterations in the cellular structure of a panel of aggressive, highly metastatic cancer cells of different tissues of origin.

Gene expression analysis comparing a panel of aggressive breast, prostate, ovarian, and melanoma cancer cell lines with their less aggressive counterparts previously revealed a commonly dysregulated signature of metabolic enzymes.

We previously identified a gene expression signature of commonly dysregulated metabolic enzymes that were heightened across a panel of highly aggressive human cancer cells, leading us to hypothesize that there was a metabolic program that supports cancer malignancy.

A recent combined differential expression and co-expression network analysis of lymphoblastoid cell lines derived from 62 patients with aggressive prostate cancer and 63 patients with non-aggressive prostate cancer identified a panel of 7 differentially expressed miRs associated with aggressive disease.

Still, these data suggest that MUC-1 may be promising to include in a panel of molecular markers to distinguish aggressive disease from indolent at diagnosis.

We measured the genetically determined outcomes of aggressive interactions, and the resulting effects on mating success, in a panel of diverse inbred lines representing both natural variation and artificially selected genotypes.

We also found that DDX3 expression is directly correlated with tumorigenesis in a panel of breast epithelial cell lines ranging from non-tumorigenic (low DDX3) to highly aggressive cancer phenotypes (high DDX3) [19].

Our study adds to this field by correlating an increasingly aggressive phenotype with an increase of metastatic efficiency in a panel of metastatic cell variants.

The methylation of H3K27 mediated by EZH2 has been implicated in the aggressive phenotype of cancer cells through the repression of a panel of tumor suppressor genes [ 24, 25].

EGF4KDEL showed receptor-specific picomolar activity against a panel of human MM cell lines and also induced an impressive decrease in tumour burden in an aggressive H2373 intraperitoneal xenograft model.

SRF expression was frequently elevated in a panel of metastatic GC cells and tissues, and high-level expression of SRF was significantly associated with a more aggressive phenotype and poor prognosis in patients with GC.