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In our previous work (Campbell et al, 2003), we found that caveolin-1 can be substituted for vascular invasion in a hazard model for time to recurrence (but not for grade or capsular invasion), suggesting that its expression is linked to vascular proliferation.
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In this paper, we develop a moral hazard model for the empirical analyses of behavioral risks.
We developed a seismic hazard model for Taiwan that integrates all available tectonic, seismicity, and seismic hazard information in the region to provide risk managers and engineers with a model they can use to estimate earthquake losses and manage seismic risk in Taiwan.
The classic way to analyze competing risks data is to study event-specific hazard rates, i.e., fitting a proportional hazard model for each event (NB, death without NB and discharge without NB) separately.
More sophisticated and computationally intensive approaches for fitting the working model could be used, such as a proportional hazard model for interval-censored data, but we suspect that would not lead to a significant reduction in the bias, which is the major concern under a situation with dependent censoring for the WKM method.
Therefore, the city planners and the seismic insurance product designers often require an empirical earthquake hazard model for a large geographical region of interest.
We fitted a Cox proportional hazard model for time to improvement for the same demographic and clinical covariates assessed in KS presentation.
We will use a Cox proportional hazard model for the analysis.
To test the hypothesis under study, we used a Cox proportional hazard model for weaning.
We fitted a Cox proportional hazard model for each gene and tested for the influence of gene expression on survival outcome.
To compare DFS and OS rates, the log-rank test was used for univariate analysis, and a Cox proportional hazard model for uni- and multivariate analyses.
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