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At present, adults are generally tested for evidence of genetic disease only if personal or family history suggests they are at increased risk for a given disorder.
Options may even exist for carrier testing, studies that determine whether an individual is at increased risk of having a child with a given disorder, even though he or she personally may never display symptoms.
In the case of genetic disease, options often exist for presymptomatic diagnosis that is, diagnosis of individuals at risk for developing a given disorder, even though at the time of diagnosis they may be clinically healthy.
The future of psychotherapy research lies in the development of easy-to-use, efficient treatments that target specific characteristics and needs of patients with a given disorder.
Thus, there is strong evidence that genes that cause a given disorder are likely to be more similarly co-expressed across diverse gene expression experiments that UGET is detecting.
Understanding the properties of each analysis method is critical in deciding whether the interpretation of pathological movements observed in a given disorder should be inferred based on the neural noise or loss of complexity hypothesis.
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For each disorder, gene-given disorder associations are derived from a PubMed search linking the given disorder name with gene names.
We will follow the cohort from a given age (where the 'at risk' for developing a given mental disorder is present), until the date of their first diagnosis for this given disorder, the date of death, the date of emigration/disappearance or 1 January 2015, whichever comes first.
The population attributable fraction of a given mental disorder is thus the proportion of mental disorder in the total study population which could be avoided if no physical, sexual, psychological violence respectively were at hand.
First, starting with a gene set associated with a given human disorder (e.g., genes associated with a given human autoimmune disorder characterized by an unknown environmental trigger), GSEA could be used to assay a number of conditions in which those genes might be differentially expressed in an appropriate model organism.
Therefore, identifying such correlated deficits or a circuit endophenotype would provide a mechanistic point of entry, enhancing both diagnosis and treatment of a given behavioral disorder.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com