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Taken together the studies described above highlight the importance of a fall in viable chondrocyte number in preventing integration of tissues and the need for a cell based strategy to enhance integration.
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Three seconds after contact, as little as 10 ug of PlyG mediated a 5,000-fold 5,000-foldn viable counts of a ~107 bacillus culture [ 8].
B. subtilis and B. stearothermophilus proved to be BIs appropriate for the evaluation of glutaraldehyde as a sterilizing agent, requiring exposure times of 300 min (5 h), to undergo a 12log10 reduction in viable spore forms.
They found that in combination with gentamicin, pulsed US applied continuously for up to 72 h at a frequency of 28.48 Hz and a maximum intensity of 0.5 W/cm resulted in a 58 69% reduction in viable E. coli biofilm on bone cement compared to the negative controls.
Hirel et al. (1996) observed in viable human keratinocytes obtained 24 50 h after death EROD and phenacetin deethylase activities (0.5 0.7 pmol/min/mg protein), which were similar as in keratinocytes from freshly obtained surgical samples.
Hirel et al. (1996) observed in viable human keratinocytes obtained 24 50 h after death NAT activities toward procainamide as substrate (around 4 pmol/min/mg protein), which were similar to those determined in keratinocytes freshly obtained from surgery.
Hirel et al. (1996) observed in viable human keratinocytes obtained 24 50 h after death GST activity toward CDNB, which was similar as in keratinocytes from freshly obtained surgical samples (around 40 nmol/min/mg protein).
Hirel et al. (1996) observed in viable human keratinocytes obtained 24 50 h after death SULT activities toward paracetamol as substrate (around 0.4 pmol/min/mg protein), which were about half compared with keratinocytes freshly obtained from surgery.
By contrast, S. enterica serovar Enteritidis and E. coli O157 H7 showed a reduction in viable cells number after 8 h exposure, and the growth inhibition was determined by counting the numbers of CFUs for the bacteria (Fig. 4A: e, f).
Here we demonstrate that in vivo deletion of the Scl +19 enhancer (Scl Δ19/Δ19 ) results in viable mice with normal Scl expression in mature hematopoietic lineages.
In addition, as demonstrated by 100 failed attempts to generate a cloned macaque in 2007, a viable pregnancy is not guaranteed.
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