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Each mucosal layer holds a distinct set of functions that are together responsible for VF immune, transport and barrier capabilities, the ability to absorb considerable impact stress, and favorable viscoelasticity for self-sustained tissue oscillation and voice production [1] [9].
GO term analysis indicates that most genes from each of the three tissues are associated with terms that are found only for that tissue, i.e., each tissue has a distinct set of functions that is associated with extended 3'UTRs and small RNAs (Additional file 1: Figure S4e and Additional Table S4).
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Each interaction type is associated with a distinct set of biological functions.
Importantly, although there are two Tor complexes in S. cerevisiae, TORC1 and TORC2, each of which regulate a distinct set of cellular functions, (Loewith et al. 2002), the SEA complex has only been established as a regulator of TORC1.
The overlap between different class combinations in terms of GO categories associated with them is very low, implying that each interaction type is intrinsic for a distinct set of cellular functions (see Additional file 4).
Note that there was a trend towards concentration of a distinct set of processes and functions in the group of proteins making up the Melampsora secretome.
Each type may have a distinct set of associated physiological, psychological and neurological functions.
How come river cities seem more robust in their syntactic structure than non-river cities, when they clearly have a distinct set of connections that are crucial for their function?
Based upon the low number of overlapping hits between each screen and the results from our GO term analyses, we conclude that each minisatellite tract is regulated by a distinct set of genes that do not share many overlapping functions or processes.
Two taste receptor neurons, ASE left (ASEL) and ASE right (ASER) are morphologically bilaterally symmetric, but they display a left/right asymmetric function and express a distinct set of chemosensory receptors (Sarin et al., 2007; Ortiz et al., 2009).
Thus, in a distinct set of BRCA-FA pathway deficient cells, loss of MMR function could synergize and contribute to tumors in FA patients.
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