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(2) Duration of the disorder: There was also a discrepancy in the duration of the disorder among patients, varying from one to ten years.
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As a result, there was a discrepancy between the duration of the predictions made in simulation and the real-world trial period.
A difference in the duration of follow-up cannot explain this discrepancy.
Part of the discrepancy may stem from differences in the duration of diabetes, type of treatment, and baseline glucose control of diabetic patients included in the studies (3– 5).
These discrepancies may be related to differences in the duration of sexual experience across studies or possible variations in the testing conditions that are known to influence behavior in the elevated plus maze [49].
The main reason for this discrepancy may be the duration of the thermodilution evaluation period.
The reason for this substantial discrepancy could be the duration of disease, rather than different genetic pathogenesis.
This discrepancy could be explained in part by differences in the duration of treatment or history of diabetes mellitus.
The discrepancy between the results of the current and our previous study [9] may be related to the differences in the duration of the delay periods used in both studies.
Additionally, the expected clinical variability in the duration of ischemia in the studies combined with the various IPC protocol may potentially be contributing to the discrepancy of results seen.
What are the main differences between cohort 1 and cohort 2 that might explain the discrepancy in vaccine duration of protection?
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