Although 111In-labelled 11B6 showed a good stability in murine serum, we investigated whether the high uptake seen in the salivary glands could be a result of indium-labelled CHX-A -DTPA, this was not CHX-A -DTPAFigure 5, left).
Figure 2 A confirms that epistatic interactions underlying growth traits follow the power-law distribution.
Figure 8 A confirms the specificity of bouton labeling using GFP-tagged synaptophysin.
This is complementary to recent findings demonstrating that SGs are cleared by autolysosomes and accumulate during the recovery, if autophagy is inhibited (a phenomenon we confirmed; Supplementary Figure S3A).
Using symmetry arguments, we grouped 3- and 4-gene motifs into buffered and non-buffered categories, which are equi-probable in a random network (confirmed by Figure 5 figure supplement 4B,E,F,H,K,L).
Figure 3(a) confirms the results.
Figure 4(a) confirms our intuition: mobility with local cooperation can help computing global properties incurring in a small overhead.
Furthermore, the punctuated cellular LipidTOX localization in Figure 5 a confirms the accumulation of LipidTOX in cellular lysosomes upon induction of PLD by 79.
The experiments shown in Figure 2(A) confirm that asm-deficient mice and wild-type control develop comparable hypercholesterolaemia when placed on atherogenic diet.
