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In contrast, the concentrations of Ba (renal medulla), Zn (renal cortex and medulla), and Mn (liver, renal medulla) were lower in cats with a CKD compared to cats without a CKD (P < 0.05).
This would yield a CKD prevalence of 4.3% for our model (57/1313) under the assumption that CKD occurred after at least 15 years of treatment.
Periodontal diseases are common bacterial plaque-induced inflammatory conditions that can respond to treatment and have been implicated as a CKD risk factor.
The CONTACT study recruited practices during a CKD management course for GPs in the eastern Netherlands.
The inclusion criteria were being a CKD patient and not receiving RRT.
The hypothesis of this study is that the estimated nephron number can predict the outcome of a CKD patient.
Timely uACR testing was better from 2009, consistent with the introduction of uACR as a CKD QOF indicator that year.
The Cd concentrations in the liver and renal cortex were higher in cats with a CKD compared to healthy cats.
We previously demonstrated that giving primary care providers access to a CKD registry does not improve guideline adherence [ 16].
Epoetin delta was well tolerated, with adverse events occurring at rates expected for a CKD patient population; no patient developed anti-erythropoietin antibodies.
Similar to our results, a CKD checklist provided to primary care physicians improved guideline adherence but not blood pressure control in a non-randomized quality improvement study [ 26].
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