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While this is sometimes the case, our data suggest that if HIV RNA is a cause of enhanced proinflammatory response to endotoxin, then by removing circulating HIV RNA, such as with ART, normal tolerance to LPS could potentially be restored.
The association of IQGAP1 with active Rac1 results in destabilisation of adherens junction as a cause of enhanced E-cadherin protein degradation.
Similar to what has been recorded in normal Leydig cells, we first found the bulk of tumour CYP19 transcripts to be PII derived, excluding promoter shift as a cause of enhanced transcription.
Expression of RANK (RANK-L receptor) was not affected and expression of c-Fms (M-CSF receptor) was only slightly elevated in osteoclasts of both mutants (data not shown), excluding elevated cytokine receptor levels as a cause of enhanced osteoclastogenesis of mutant cells.
The production of ROS associated with lipid accumulation is a cause of enhanced risk of damage and disease progression to necroinflammation, steatohepatitis, fibrosis, cirrhosis, and eventually hepatocellular carcinoma [ 4, 5] and of injuries during ischemia/reperfusion of donor organs for transplantation [ 6, 7].
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Whereas Pilon-Smit et al. (1999) [ 48] concluded that selenate tolerance was due to increased volatilization of Se in the ATP sulfurylase over-producing plants, we propose that the efficient reduction of selenate is the primary cause of enhanced Se resistance.
On the one hand, increasing the number of distribution centers is a cause to enhance the service levels and reducing the transportation costs from the distribution centers to the customers.
Increasing and worthwhile attention has been paid to applying existing international law to the cause of enhancing global cybersecurity.
Rep. Chip Roy (R-Tex). said the bill "may not actually advance the cause" of enhancing employment opportunities for those who have been convicted.
Thus, loss of aortic VASP protein appears to be a consequence rather than a cause of endothelial dysfunction and may reflect enhanced proteasomal activity of ATGL-deficient aortas.
Thus, increased formation of HNE and MDA in neurons with N-truncated Aβ deposits may enhance the formation of reactive oxygen species in mitochondria and may be the cause of, or enhance an existing mitochondrial dysfunction in autism and dup(15)/autism.
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