We recently delineated that allocation of CAEN to the brain and muscles happens mainly during daytime, while allocation of CAEN to the immune/repair systems happens at night [ 70].
There were 13 difficulties delineated that are categorized and presented in Table 3.
From these definitions, targets for subjective QOL interventions could be delineated that would have cross-cultural relevance.
A critical region was delineated that gave boundaries for breakpoints of X-autosome translocations associated with ovarian failure.
Abdel-Wahab et al. delineated that total 3 DNMT3A-positive cases in 46 primary MF patients were also found to have cooccurring JAK2V617F mutation [ 39].
As such, a set of highly reliable interactions could be delineated that can be further employed in both computational and experimental studies.
Up to four subtypes of FTLD-U have been delineated that are based on the distribution of NCIs, dystrophic neurites and the presence of neuronal intranuclear inclusions (NIIs) (8– 10).
We delineated miRNAs that showed significant association with recurrence.
In addition, we delineated the mechanism that regulates MCP-1 secretion in AECII, in which we found that Lyn, a critical Src family member, can increase MCP-1 secretion by activating the NF-κB pathway.
Using this approach, we delineated ubiquitin mutations that selectively impacted the ubiquitin-E1 edge.
