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This uptake was receptor-mediated.
This uptake pattern is shown on Fig. 2b.
This uptake is enabled by carbonations of hydration products in hardened cement paste.
This uptake mechanism involves macrophages in snatching up CNP from blood vessels.
This uptake mechanism makes folic acid also attractive for FR-targeted chemotherapy [16].
This uptake is attributed to increased glycolysis in activated inflammatory cells, mainly macrophages [81].
This uptake pattern appeared rapidly (one month) and disappeared after sorafenib withdrawal.
This uptake is further enhanced when the AuNPs are capped with proteins.
This uptake remained stable for 4h p.i. and declined to >2% ID/g at 24h p.i.
This uptake decreases as the dose rate is increased and would lead to a loss of wear resistant properties.
This uptake impacted the Wnt signaling cascade, resulting in an elevation of β-catenin levels due to GSK-3β inhibition.
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