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Our results showed that SRC measures were vulnerable to faking.
Figure 4 indicates that SRC patients are significantly younger than the non-sport related concussion patients.
These data suggest that Src kinases can modulate IL-1 production by a post-transcriptional mechanism.
Our previous results show that src can activate CKII, leading to direct downregulation of SLK (32).
Our data suggested that Src may also regulate survival pathways that block apoptosis in hypoxia.
One possible explanation for this finding is that SRC activates a phosphatase that targets these proteins.
Salvi et al reported that Src is located in mitochondria in rat brain [26].
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These results suggest that SRC-1 and KIN-22 play distinct roles under the control of CSK-1.
These results indicate that SRC-3 can suppress inflammatory response.
These demonstrate that SRC-LatLRR is effective and efficient for tumor classification.
These results indicate that SRC-3 inhibits cytokine production at both protein and mRNA levels.
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