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Therefore, we found that MPT played an important role in Pb-induced mitochondrial damage and, ultimately, cell death.
It was established by the X-ray analysis that MPT generated ferrite-austenite steels (35, 45, 40Kh steels) and ferrite-cementite (U8, ShKh15 steels) structure.
The X-ray analysis gives a possibility to establish that MPT generates the ferrite-austenite NCS on the steel surface independently of coolant type.
The production network measurements proved that MPT is capable for super-aggregation (i.e. the aggregated throughput is higher than the algebraic sum of the throughput of the paths).
It should be noted that MPT has some advantages over the other methods of severe plastic deformation: besides achieving NCS, the structural-phase transformation and saturation of the surface layer with alloying elements are realized under this treatment [6 8].
Besides, it should be taken into consideration that MPT of metal with higher carbon content (steel 45) increases the quantity of retained austenite and this can have the influence on corrosion processes of the metal too.
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It should be noted that the MPT effect became more pronounced with increase of carbon content in the steels.
Together these results suggest that only MPT were able to bind and activate monocytes to produce IL-1β.
We hypothesized that primary MPT cells, lacking one or other of the α-isoforms of AMPK, should be more susceptible to apoptosis induced by ATP depletion than primary MPT cells with both α-isoforms intact.
Mononuclear enzymes contain a cofactor that comprises metallopterin (MPT) or some of its nucleotide variants, each of which is coordinated to Mo or W with an enedithiolene motif.
It should be noted that formed during MPT oxides FeO and Fe3O4, being secondary structures could also decrease wear by the way of relieve running-in of the friction pairs [16].
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