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This suggests that MEs are only half-way in becoming self-governing units (Meyer, et al., 2017).
Statistical analysis of the results showed that MES removal did not affect cell growth and ethanol production (P > 0.05).
Previous studies indicated that MES and HEPES do not significantly interfere with Ni and Zn sorption at mineral-solution interfaces [30, 31].
Our recent study demonstrated for the first time that MEs significantly contribute to cellular NADPH generation and are vital for tumor growth (Jiang et al., 2013a).
Our analysis reveals that MES are acknowledged, but their recognition is partial and limited to the services which are already captured by market mechanisms.
In order to achieve the action of the operator (5) in the space of essentially bounded functions L ∞, we have to assume that mes { t : g i ( t ) = c } = 0 for every constant c.
Similar(14)
Moreover, previous studies have shown that Mes-NaOH buffer alone failed to trigger plant defense signaling [ 3, 6, 10, 11, 32].
The aberrant presence in mes-4 PGCs of hyperphosphorylated Pol II, H3K4me and MET-1-dependent H3K36 methylation supports a conclusion that mes-4 mutant PGCs are precociously transcriptionally engaged.
The loss of this signal in mes-4 embryos suggests that MES-4 is either essential for maintaining pre-existing H3K36 methylation in embryonic chromatin through cell divisions or must provide this marker de novo after replication-coupled depletion.
Transcript profiling of dissected mutant germlines revealed that MES-2/3/6 and MES-4 cooperate to promote the expression of germline genes and repress the X chromosomes and somatic genes.
Our analyses indicate that MES-4 and DRM cobind many germline-expressed genes.
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