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Since transcripts of AK1 and AK5 increase with cardiogenesis and AK2 is the predominant mitochondrial isoform, the AMP-generating adenylate kinase system was targeted using a combination of AK1-AK2-AK5 AK1-AK2-AK5 AK1-AK2-AK5
Since transcripts expressed in RPCs would not be expected to disappear immediately, it was predicted that some cells would possess profiles containing genes expressed in one or more neuronal cell types, together with RPC genes that are in the process of being downregulated.
Since transcripts can be impacted differently, each transcript is listed.
Since transcripts that originated from AS often hold high similarity in sequence, it is likely that problematic assignment in gene orthologs would be reported using sequence-based pipelines.
Since transcripts for these probe sets do not appear to be present above background, it seems reasonable to assume that they are not differentially expressed.
Since transcripts from different miRNA genes can be processed into the same mature miRNA, target sites for closely related miRNA genes were identified with the same screening sequence.
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Furthermore –again similar to Arabidopsis roots– increased PSY seems sufficient to explain heightened carotenoid accumulation, since transcript levels of other carotenogenic genes do not increase in orange as opposed to white-rooted carrots [32].
Although none of these maize populations is fully resolved by markers, achievement of appropriately high marker density will fade as a major limitation, since transcript-derived markers have been shown to be reliable for generation of thousands of new markers per experiment [7], and suitable transcript profiling platforms exist for at least 14 crops as well as for all animal model systems.
Moreover, Zambelli et al. suggest that the orthology relationship should be considered at the transcript level, since transcript isoforms are the individuals subject to protein functions [ 13].
Since transcript-tion and translation are coupled in mitochondria [ 24- 26], these changes probably work together to mediate the increase in OXPHOS complex abundance in tor1 Δ strains.
Furthermore, since transcript levels of AtPNP-A were reduced beyond detection limits in senescing leaves in the nahG mutant this induction appears to be SA-dependent.
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