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One of the factors involved in epigenetic changes during in vitro conditions is the exposure to growth regulators, which are widely used in plant tissue culture [ 34, 35] to promote multiplication and growth.
Tissue culture is an alternative approach and various methods have already been developed to promote multiplication of Dendrobium (Arditti and Ernst 1993; Kumaria and Tandon 2001; Dohling et al. 2007; Zhao et al. 2007; Luo et al. 2008; Chugh et al. 2009; Paul et al. 2012).
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The increased free volumes in the glass matrix after tensile pre-deformation contribute to the decrease of the Young's modulus of the glass matrix and lead to the increase in the stress concentration, promoting multiplication of shear bands.
Numerous viruses are already known to induce modifications of the ER to promote their multiplication.
Both viral proteins prevent premature insect cell death and thereby promote virus multiplication.
In combination, virulence factors promote the multiplication of bacilli after invading the human organism.
Research has been undertaken in order to find out the factors which promote uncontrolled multiplication of cells and how cancer genes affect cell signaling, chromatin, and epigenomic regulation and RNA splicing, protein homeostasis, metabolism, and lineage maturation [ 4– 6].
However, WSSV is a systemic virus that infects most shrimp tissues and organs, and it is logical to assume that in different cell types, the virus would be likely to modulate the expression of different host genes in order to promote its multiplication in the correspondingly different cellular contexts.
Although Group A streptococci were thought to be the sole cause of Fournier's gangrene [ 15], subsequent clinical series have emphasized the polymicrobial nature of the disease [ 1, 16], which is hypothesized to synergize enzyme production and promote rapid multiplication and spread of infection [ 1].
Taken together, these data reveal that XopD is a unique virulence factor in Xcv that alters host transcription, promotes pathogen multiplication, and delays the onset of leaf chlorosis and necrosis.
The reason is that the ECP makes the crystal nuclei formed on the wall of the mould fall off and move freely in the molten metal, promoting the multiplication of crystal nuclei.
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