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The lack of transmigration with ethanol treatment was corroborated by a failure to see increases in supportive mechanisms.
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Exposure to ethanol eliminated these interactions suggesting an early block of transmigration.
Another hymn to the wonders of transmigration.
After the 1990s, however, the rate of transmigration slowed considerably.
The speculations on time reflect the doctrine of the eternal return in the philosophy of transmigration.
Two different routes of transmigration are known: paracellular at endothelial borders (70 90 %) [14] or transcellular.
Together these observations suggest that the general suppression of transmigration by ethanol was most likely due to a failure at early stages of transmigration.
The measurements showed a decrease both of transmigration and proteinuria as marker for microcirculatory damage.
Indeed the A375 cell line, secreting GM-CSF, has the highest rate of transmigration.
This suggests that no dominant effect of transmigration is present in the context of the macrocirculation.
22 Chemokines are important mediators of transmigration of leukocytes across the blood brain barrier.
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