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Numbers represent experimental pressure in GPa.
Collectively, these three variables accounted for 20% of the variance in GPA.
Other respiratory presentations include pulmonary infiltrates and nodules, the latter being observed predominantly in GPA [6].
This was unchanged in GPA (median 95%%).
Thus, NCEE score explained 6.8% of the variation in GPA.
Neutrophils play the primary pathogenic role in GPA [ 13– 15].
NK cells were not detectable in GPA granulomas.
T cells are abundant in inflammatory lesions in GPA.
Th17 subsets co-producing IFNγ or IL-10 are significantly increased in GPA.
Importantly, whether NK cells can be found in GPA tissue lesions is currently unknown.
The frequencies of CCR4+ and CCR6+ TVEM were also significantly increased in GPA.
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CEO of Professional Science Editing for Scientists @ prosciediting.com